Hemostasis, Thrombosis & Coagulation¶
26 entries, newest first.
- 0036 — What data supports the use of sirolimus in refractory ITP?
- 0034 — Should we perform bone marrow biopsies in patients with ITP?
- 0033 — What is the influence of race and ethnicity on the diagnosis of VWD?
- 0032 — How does congenital dysfibrinogenemia present in women?
- 0031 — What are the mechanisms of acquired von Willebrand syndrome?
- 0030 — What is considered a safe threshold for neuraxial anesthesia in patients with bleeding disorders?
- 0028 — Should patients with antiphospholipid antibody syndrome without thrombotic or obstetric complications receive aspirin for primary prophylaxis?
- 0027 — Can emicizumab be used to treat patients with severe type 3 VWD?
- 0026 — Can persons with hemophilia receive antithrombotic therapy?
- 0021 — How do you diagnose bleeding disorders of unknown cause (BDUC)?
- 0018 — Describe the natural history, definition, diagnosis, investigations, and management of patients with confirmed and suspected VWD.
- 0016 — What treatment options are available for patients with FVII deficiency?
- 0015 — What is your approach to the use of porcine FVIII in acquired hemophilia A?
- 0014 — How do you choose between steroids, cyclophosphamide, and rituximab for inhibitor elimination in acquired hemophilia A?
- 0013 — What is the correlation between factor VIII levels and bleeding in carriers of hemophilia A?
- 0012 — What is the difference between hemophilia carriers and hemophilia?
- 0011 — Can you use TPO-RAs in patients with ITP and positive APLAs?
- 0010 — Can patients with persistent or chronic ITP eventually stop treatment with TPO-RAs?
- 0008 — Is IVIG use in ITP associated with increased risk of thrombosis?
- 0007 — What tests for qualitative platelet function defects do we have available, and how can they influence management in clinic?
- 0006 — In frail elderly patients who are stable on warfarin for stroke prevention from atrial fibrillation, can you switch them to a DOAC? And does that lead to harm?
- 0005 — How can someone who has a low factor VIII level of around 40% be considered to possibly have type 2N VWD?
- 0004 — What is the F8 gene duplication (FVIII Padua)?
- 0003 — Can we use Altuviiio (efanesoctocog alfa) for patients who have a history of inhibitors and underwent ITI?
- 0002 — Explain why there is discordance in FVIII activity after AAV gene therapy between one-stage (OS) and chromogenic substrate (CS) assays.
- 0001 — Why is there a discrepancy between clot-based one-stage assays and chromogenic assays in patients who have undergone gene therapy for hemophilia B?